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may involve the glossopharyngeal nerve. Fainting as a manifestation of vagoglossopharyngeal neuralgia is described on page 325. The same drugs that have been found helpful in the treatment of tic douloureux may be used to treat glossopharyngeal neuralgia, but their ef cacy has been dif cult to judge. Regarding vascular compression of the nerve, Resnick and colleagues have reported the results of microvascular decompression of the ninth nerve in 40 patients; in 32 of these, relief of symptoms was complete and was sustained during an average follow-up of 4 years; 3 patients remained with permanent weakness of structures thought to be innervated by the ninth nerve. A similar high rate of success has been achieved by others. If syncope is associated with the pain, it can be expected to cease with abolition of the attacks of pain. Syncope can also occur when the ninth nerve is involved by tumors of the parapharyngeal space; most of these are squamous cell carcinomas and both the ninth and tenth nerves are implicated. Section of rootlets of the ninth nerve has reportedly reduced or abolished the episodes of fainting in these cases.

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This part of the cerebrum, lying behind the central sulcus and above the sylvian ssure, is the least well demarcated (Fig 22-7) Its posterior boundary, where it merges with the occipital lobe, is obscure, as is part of the inferior boundary, where it merges with the temporal lobe On its medial side, the parieto-occipital sulcus marks the posterior border, which is completed by extending the line of the sulcus downward to the preoccipital notch on the inferior border of the hemisphere Within the parietal lobe, there are two important sulci: the postcentral sulcus, which forms the posterior boundary of the somesthetic cortex, and the interparietal sulcus, which runs anteroposteriorly from the middle of the posterior central sulcus and separates the mass of the parietal lobe into superior and inferior lobules (Figs 22-2 and 22-7).

Brain Lang 6:82, 1975 ROSS ED: The Aprosodias, in Feinberg TE, Farah MJ (eds): Behavioral Neurology and Neuropsychology New York, McGraw-Hill, 1997, pp 699 717 SUBIRANA A: Handedness and cerebral dominance, in Vinken PJ, Bruyn GW (eds): Handbook of Clinical Neurology Vol 4: Disorders of Speech, Perception and Symbolic Behavior Amsterdam, North-Holland, 1969, pp 284 292 SYMONDS C: Aphasia J Neurol Neurosurg Psychiatry 16:1, 1953 WENDER D: Aphasic victim as investigatior Arch Neurol 46:91, 1989 WERNICKE C: Der Aphasische Symptomkomplex Breslau, Germany, Kohn & Weigert, 1874 English translation by Eggert GH: Wernicke Works on Aphasia: A Source Book and Review The Hague, Mouton, 1977 WERTZ RT, WEISS DG, ATEN JL, et al: Comparison of clinic, home, and deferred language treatment for aphasia: A Veterans Administration Cooperative study Arch Neurol 43:653, 1986 WILLMES K, POECK K: To what extent can aphasic syndromes be localized Brain 116:1527, 1993 WILSON SAK: Aphasia.

A stored procedure is defined in a query using the Create Procedure statement. You can enter the query directly into an interactive software tool provided by the DBMS such as the Microsoft SQL Server Enterprise Manager or Query Analyzer. Alternatively, you can execute the query from your application by using the ExecuteNonQuery() function, which is illustrated in examples throughout this chapter. It is important to understand that not all DBMSs support stored procedures, and therefore, you'll need to check with the DBMS manufacturer before incorporating stored procedures in your application. The Create Procedure statement requires a unique name, SQL statements that are to execute when the stored procedure is called, and a return value if required by your application. You'll find that some stored procedures, such as those used to insert a new row into the database, don't require a return value, while others, such as procedures for counting the number of customers, do require one. Let's take a look at a simple stored procedure that will count the number of customers there are in the custcontact table. We'll call this HowManyCustomers:

The inferior parietal lobule is composed of the supramarginal gyrus (Brodmann s area 40) and the angular gyrus (area 39) The architecture of the postcentral convolution is typical of all primary receptive areas (homotypical granular cortex) The rest of the parietal lobe resembles the association cortex, both unimodal and heteromodal, of the frontal and temporal lobes The superior and inferior parietal lobules and adjacent parts of the temporal and occipital lobes are relatively much larger in humans than in any of the other primates and are relatively slow in attaining their fully functional state (beyond the seventh year of age) This area of heteromodal cortex has large ber connections with the frontal, occipital, and temporal lobes of the same hemisphere and, through the middle part of the corpus callosum, with corresponding parts of the opposite hemisphere.

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